Arrival of excitation at the left ventricular apical endocardium in Wolff-Parkinson-White syndrome type B
Identifieur interne : 000124 ( Main/Corpus ); précédent : 000123; suivant : 000125Arrival of excitation at the left ventricular apical endocardium in Wolff-Parkinson-White syndrome type B
Auteurs : Otto L. Garcia ; Agustin Castellanos ; Maria C. Vagueiro ; Robert J. Myerburg ; Henry GelbandSource :
- Journal of Electrocardiology [ 0022-0736 ] ; 1982.
Abstract
Electrograms were recorded from the His bundle area, right ventricular apex, right ventricular inflow tract, and left ventricular apical endocardium, in four patients (aged, 1, 1, 1.5, and 16 years) with Wolff-Parkinson-White syndrome type B. In beats without preexcitation: (a) delayed activation of the right ventricular inflow tract reflected the occurrence of a conduction disturbance through the “distal” or “peripheral” ramifications of the right bundle branch; and (b) the slightly earlier activation of the left ventricular apical endocardium (in reference to the right ventricular apex) may have been due to an earlier emergence from the divisions of the, left bundle branch, presumably due to the greater length of the right bundle branch. In beats with preexcitation: (a) the “incomplete” right bundle branch block pattern was concealed because the right ventricular inflow tract was activated before the right ventricular apex; (b) the deltaright ventricular apical intervals were shorter than those of adults with Wolff-Parkinson-White type B; and (c) arrival of excitation at the left ventricular endocardium was a function, either of the impulse emerging from the left bundle branch, or of that propagating from the preexcited site. Therefore, the delta-left ventricular apical endocardial intervals were considered to have represented conduction time from preexcited region to endocardium of left ventricle only when it could be proven that the conduction time (from atrial site of origin to left ventricular apical endocardium) was shorter through the right sided accessory pathway than through the normal pathway. This study suggests that some beats, which may be interpreted as representing “pure” Wolff-Parkinson-White type B complexes from epicardial maps, may in reality be “fusion” complexes.
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DOI: 10.1016/S0022-0736(82)80012-5
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<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Arrival of excitation at the left ventricular apical endocardium in Wolff-Parkinson-White syndrome type B</title><author><name sortKey="Garcia, Otto L" sort="Garcia, Otto L" uniqKey="Garcia O" first="Otto L." last="Garcia">Otto L. Garcia</name></author><author><name sortKey="Castellanos, Agustin" sort="Castellanos, Agustin" uniqKey="Castellanos A" first="Agustin" last="Castellanos">Agustin Castellanos</name></author><author><name sortKey="Vagueiro, Maria C" sort="Vagueiro, Maria C" uniqKey="Vagueiro M" first="Maria C." last="Vagueiro">Maria C. Vagueiro</name></author><author><name sortKey="Myerburg, Robert J" sort="Myerburg, Robert J" uniqKey="Myerburg R" first="Robert J." last="Myerburg">Robert J. Myerburg</name></author><author><name sortKey="Gelband, Henry" sort="Gelband, Henry" uniqKey="Gelband H" first="Henry" last="Gelband">Henry Gelband</name></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:8849C84861D8758EDCCBACC7D0692B08F77FF1C1</idno><date when="1982" year="1982">1982</date><idno type="doi">10.1016/S0022-0736(82)80012-5</idno><idno type="url">https://api.istex.fr/document/8849C84861D8758EDCCBACC7D0692B08F77FF1C1/fulltext/pdf</idno><idno type="wicri:Area/Main/Corpus">000124</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">Arrival of excitation at the left ventricular apical endocardium in Wolff-Parkinson-White syndrome type B</title><author><name sortKey="Garcia, Otto L" sort="Garcia, Otto L" uniqKey="Garcia O" first="Otto L." last="Garcia">Otto L. Garcia</name></author><author><name sortKey="Castellanos, Agustin" sort="Castellanos, Agustin" uniqKey="Castellanos A" first="Agustin" last="Castellanos">Agustin Castellanos</name></author><author><name sortKey="Vagueiro, Maria C" sort="Vagueiro, Maria C" uniqKey="Vagueiro M" first="Maria C." last="Vagueiro">Maria C. Vagueiro</name></author><author><name sortKey="Myerburg, Robert J" sort="Myerburg, Robert J" uniqKey="Myerburg R" first="Robert J." last="Myerburg">Robert J. Myerburg</name></author><author><name sortKey="Gelband, Henry" sort="Gelband, Henry" uniqKey="Gelband H" first="Henry" last="Gelband">Henry Gelband</name></author></analytic><monogr></monogr><series><title level="j">Journal of Electrocardiology</title><title level="j" type="abbrev">YJELC</title><idno type="ISSN">0022-0736</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1982">1982</date><biblScope unit="volume">15</biblScope><biblScope unit="issue">2</biblScope><biblScope unit="page" from="165">165</biblScope><biblScope unit="page" to="172">172</biblScope></imprint><idno type="ISSN">0022-0736</idno></series><idno type="istex">8849C84861D8758EDCCBACC7D0692B08F77FF1C1</idno><idno type="DOI">10.1016/S0022-0736(82)80012-5</idno><idno type="PII">S0022-0736(82)80012-5</idno><idno type="ArticleID">82800125</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0022-0736</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Electrograms were recorded from the His bundle area, right ventricular apex, right ventricular inflow tract, and left ventricular apical endocardium, in four patients (aged, 1, 1, 1.5, and 16 years) with Wolff-Parkinson-White syndrome type B. In beats without preexcitation: (a) delayed activation of the right ventricular inflow tract reflected the occurrence of a conduction disturbance through the “distal” or “peripheral” ramifications of the right bundle branch; and (b) the slightly earlier activation of the left ventricular apical endocardium (in reference to the right ventricular apex) may have been due to an earlier emergence from the divisions of the, left bundle branch, presumably due to the greater length of the right bundle branch. In beats with preexcitation: (a) the “incomplete” right bundle branch block pattern was concealed because the right ventricular inflow tract was activated before the right ventricular apex; (b) the deltaright ventricular apical intervals were shorter than those of adults with Wolff-Parkinson-White type B; and (c) arrival of excitation at the left ventricular endocardium was a function, either of the impulse emerging from the left bundle branch, or of that propagating from the preexcited site. Therefore, the delta-left ventricular apical endocardial intervals were considered to have represented conduction time from preexcited region to endocardium of left ventricle only when it could be proven that the conduction time (from atrial site of origin to left ventricular apical endocardium) was shorter through the right sided accessory pathway than through the normal pathway. This study suggests that some beats, which may be interpreted as representing “pure” Wolff-Parkinson-White type B complexes from epicardial maps, may in reality be “fusion” complexes.</div></front></TEI><istex><corpusName>elsevier</corpusName><author><json:item><name>Otto L. Garcia M.D.</name></json:item><json:item><name>Agustin Castellanos M.D.</name></json:item><json:item><name>Maria C. Vagueiro M.D.</name></json:item><json:item><name>Robert J. Myerburg M.D.</name></json:item><json:item><name>Henry Gelband M.D.</name></json:item></author><articleId><json:string>82800125</json:string></articleId><language><json:string>eng</json:string></language><abstract>Electrograms were recorded from the His bundle area, right ventricular apex, right ventricular inflow tract, and left ventricular apical endocardium, in four patients (aged, 1, 1, 1.5, and 16 years) with Wolff-Parkinson-White syndrome type B. In beats without preexcitation: (a) delayed activation of the right ventricular inflow tract reflected the occurrence of a conduction disturbance through the “distal” or “peripheral” ramifications of the right bundle branch; and (b) the slightly earlier activation of the left ventricular apical endocardium (in reference to the right ventricular apex) may have been due to an earlier emergence from the divisions of the, left bundle branch, presumably due to the greater length of the right bundle branch. In beats with preexcitation: (a) the “incomplete” right bundle branch block pattern was concealed because the right ventricular inflow tract was activated before the right ventricular apex; (b) the deltaright ventricular apical intervals were shorter than those of adults with Wolff-Parkinson-White type B; and (c) arrival of excitation at the left ventricular endocardium was a function, either of the impulse emerging from the left bundle branch, or of that propagating from the preexcited site. Therefore, the delta-left ventricular apical endocardial intervals were considered to have represented conduction time from preexcited region to endocardium of left ventricle only when it could be proven that the conduction time (from atrial site of origin to left ventricular apical endocardium) was shorter through the right sided accessory pathway than through the normal pathway. This study suggests that some beats, which may be interpreted as representing “pure” Wolff-Parkinson-White type B complexes from epicardial maps, may in reality be “fusion” complexes.</abstract><qualityIndicators><score>7.377</score><pdfVersion>1.3</pdfVersion><pdfPageSize>612 x 864 pts</pdfPageSize><refBibsNative>true</refBibsNative><keywordCount>0</keywordCount><abstractCharCount>1815</abstractCharCount><pdfWordCount>4377</pdfWordCount><pdfCharCount>24279</pdfCharCount><pdfPageCount>8</pdfPageCount><abstractWordCount>267</abstractWordCount></qualityIndicators><title>Arrival of excitation at the left ventricular apical endocardium in Wolff-Parkinson-White syndrome type B</title><pii><json:string>S0022-0736(82)80012-5</json:string></pii><genre><json:string>research-article</json:string></genre><host><volume>15</volume><pii><json:string>S0022-0736(82)X8001-5</json:string></pii><pages><last>172</last><first>165</first></pages><issn><json:string>0022-0736</json:string></issn><issue>2</issue><genre><json:string>Journal</json:string></genre><language><json:string>unknown</json:string></language><title>Journal of Electrocardiology</title><publicationDate>1982</publicationDate></host><categories><wos><json:string>CARDIAC & CARDIOVASCULAR SYSTEMS</json:string></wos></categories><publicationDate>1982</publicationDate><copyrightDate>1982</copyrightDate><doi><json:string>10.1016/S0022-0736(82)80012-5</json:string></doi><id>8849C84861D8758EDCCBACC7D0692B08F77FF1C1</id><fulltext><json:item><original>true</original><mimetype>application/pdf</mimetype><extension>pdf</extension><uri>https://api.istex.fr/document/8849C84861D8758EDCCBACC7D0692B08F77FF1C1/fulltext/pdf</uri></json:item><json:item><original>true</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/8849C84861D8758EDCCBACC7D0692B08F77FF1C1/fulltext/txt</uri></json:item><json:item><original>false</original><mimetype>application/zip</mimetype><extension>zip</extension><uri>https://api.istex.fr/document/8849C84861D8758EDCCBACC7D0692B08F77FF1C1/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/8849C84861D8758EDCCBACC7D0692B08F77FF1C1/fulltext/tei"><teiHeader><fileDesc><titleStmt><title level="a" type="main" xml:lang="en">Arrival of excitation at the left ventricular apical endocardium in Wolff-Parkinson-White syndrome type B</title></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>ELSEVIER</publisher><availability><p>ELSEVIER</p></availability><date>1982</date></publicationStmt><notesStmt><note>The costs of publication of this article were defrayed in part bythe payment of page charges. This article must therefore be hereby marked “advertisement” in accordance with 18 U.S.C. § 1734 solely to indicate this fact.</note></notesStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Arrival of excitation at the left ventricular apical endocardium in Wolff-Parkinson-White syndrome type B</title><author><persName><forename type="first">Otto L.</forename><surname>Garcia</surname></persName><roleName type="degree">M.D.</roleName><note type="biography">Associate Professor of Pediatrics</note><note type="biography">From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</note><affiliation>Associate Professor of Pediatrics</affiliation><affiliation>From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</affiliation></author><author><persName><forename type="first">Agustin</forename><surname>Castellanos</surname></persName><roleName type="degree">M.D.</roleName><note type="biography">Professor of Medicine</note><note type="biography">From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</note><affiliation>Professor of Medicine</affiliation><affiliation>From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</affiliation></author><author><persName><forename type="first">Maria C.</forename><surname>Vagueiro</surname></persName><roleName type="degree">M.D.</roleName><note type="biography">Associate, Professor of Medicine</note><note type="biography">From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</note><affiliation>Associate, Professor of Medicine</affiliation><affiliation>From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</affiliation></author><author><persName><forename type="first">Robert J.</forename><surname>Myerburg</surname></persName><roleName type="degree">M.D.</roleName><note type="biography">Professor of Medicine</note><note type="biography">From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</note><affiliation>Professor of Medicine</affiliation><affiliation>From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</affiliation></author><author><persName><forename type="first">Henry</forename><surname>Gelband</surname></persName><roleName type="degree">M.D.</roleName><note type="biography">Professor of Pediatrics</note><note type="biography">From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</note><affiliation>Professor of Pediatrics</affiliation><affiliation>From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</affiliation></author></analytic><monogr><title level="j">Journal of Electrocardiology</title><title level="j" type="abbrev">YJELC</title><idno type="pISSN">0022-0736</idno><idno type="PII">S0022-0736(82)X8001-5</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1982"></date><biblScope unit="volume">15</biblScope><biblScope unit="issue">2</biblScope><biblScope unit="page" from="165">165</biblScope><biblScope unit="page" to="172">172</biblScope></imprint></monogr><idno type="istex">8849C84861D8758EDCCBACC7D0692B08F77FF1C1</idno><idno type="DOI">10.1016/S0022-0736(82)80012-5</idno><idno type="PII">S0022-0736(82)80012-5</idno><idno type="ArticleID">82800125</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1982</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>Electrograms were recorded from the His bundle area, right ventricular apex, right ventricular inflow tract, and left ventricular apical endocardium, in four patients (aged, 1, 1, 1.5, and 16 years) with Wolff-Parkinson-White syndrome type B. In beats without preexcitation: (a) delayed activation of the right ventricular inflow tract reflected the occurrence of a conduction disturbance through the “distal” or “peripheral” ramifications of the right bundle branch; and (b) the slightly earlier activation of the left ventricular apical endocardium (in reference to the right ventricular apex) may have been due to an earlier emergence from the divisions of the, left bundle branch, presumably due to the greater length of the right bundle branch. In beats with preexcitation: (a) the “incomplete” right bundle branch block pattern was concealed because the right ventricular inflow tract was activated before the right ventricular apex; (b) the deltaright ventricular apical intervals were shorter than those of adults with Wolff-Parkinson-White type B; and (c) arrival of excitation at the left ventricular endocardium was a function, either of the impulse emerging from the left bundle branch, or of that propagating from the preexcited site. Therefore, the delta-left ventricular apical endocardial intervals were considered to have represented conduction time from preexcited region to endocardium of left ventricle only when it could be proven that the conduction time (from atrial site of origin to left ventricular apical endocardium) was shorter through the right sided accessory pathway than through the normal pathway. This study suggests that some beats, which may be interpreted as representing “pure” Wolff-Parkinson-White type B complexes from epicardial maps, may in reality be “fusion” complexes.</p></abstract></profileDesc><revisionDesc><change when="1982">Published</change></revisionDesc></teiHeader></istex:fulltextTEI></fulltext><metadata><istex:metadataXml wicri:clean="Elsevier doc found" wicri:toSee="Elsevier, no converted or simple article"><istex:xmlDeclaration>version="1.0" encoding="utf-8"</istex:xmlDeclaration><istex:docType PUBLIC="-//ES//DTD journal article DTD version 5.0.1//EN//XML" URI="art501.dtd" name="istex:docType"></istex:docType><istex:document><article version="5.0" xml:lang="en" docsubtype="fla"><item-info><jid>YJELC</jid><aid>82800125</aid><ce:pii>S0022-0736(82)80012-5</ce:pii><ce:doi>10.1016/S0022-0736(82)80012-5</ce:doi><ce:copyright type="unknown" year="1982">Research in Electrocardiology, Inc.</ce:copyright></item-info><head><ce:article-footnote><ce:label>2</ce:label><ce:note-para>The costs of publication of this article were defrayed in part bythe payment of page charges. This article must therefore be hereby marked “<ce:italic>advertisement</ce:italic>” in accordance with 18 U.S.C. § 1734 solely to indicate this fact.</ce:note-para></ce:article-footnote><ce:title>Arrival of excitation at the left ventricular apical endocardium in Wolff-Parkinson-White syndrome type B</ce:title><ce:author-group><ce:author><ce:degrees>M.D.</ce:degrees><ce:given-name>Otto L.</ce:given-name><ce:surname>Garcia</ce:surname><ce:roles>Associate Professor of Pediatrics</ce:roles><ce:cross-ref refid="fn1"><ce:sup>1</ce:sup></ce:cross-ref></ce:author><ce:author><ce:degrees>M.D.</ce:degrees><ce:given-name>Agustin</ce:given-name><ce:surname>Castellanos</ce:surname><ce:roles>Professor of Medicine</ce:roles><ce:cross-ref refid="fn1"><ce:sup>1</ce:sup></ce:cross-ref><ce:cross-ref refid="cor1"><ce:sup>3</ce:sup></ce:cross-ref></ce:author><ce:author><ce:degrees>M.D.</ce:degrees><ce:given-name>Maria C.</ce:given-name><ce:surname>Vagueiro</ce:surname><ce:roles>Associate, Professor of Medicine</ce:roles><ce:cross-ref refid="fn1"><ce:sup>1</ce:sup></ce:cross-ref></ce:author><ce:author><ce:degrees>M.D.</ce:degrees><ce:given-name>Robert J.</ce:given-name><ce:surname>Myerburg</ce:surname><ce:roles>Professor of Medicine</ce:roles><ce:cross-ref refid="fn1"><ce:sup>1</ce:sup></ce:cross-ref></ce:author><ce:author><ce:degrees>M.D.</ce:degrees><ce:given-name>Henry</ce:given-name><ce:surname>Gelband</ce:surname><ce:roles>Professor of Pediatrics</ce:roles><ce:cross-ref refid="fn1"><ce:sup>1</ce:sup></ce:cross-ref></ce:author><ce:correspondence id="cor1"><ce:label>3</ce:label><ce:text>Reprint requests to: Agustin Castellanos, M.D., Division ofCardiology (D-39), University of Miami School of Medicine, P.O. Box 016960, Miami, Florida, 33101.</ce:text></ce:correspondence><ce:footnote id="fn1"><ce:label>1</ce:label><ce:note-para>From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</ce:note-para></ce:footnote></ce:author-group><ce:abstract id="ab1" class="author" xml:lang="en"><ce:section-title>Summary</ce:section-title><ce:abstract-sec><ce:simple-para>Electrograms were recorded from the His bundle area, right ventricular apex, right ventricular inflow tract, and left ventricular apical endocardium, in four patients (aged, 1, 1, 1.5, and 16 years) with Wolff-Parkinson-White syndrome type B. In beats without preexcitation: (a) delayed activation of the right ventricular inflow tract reflected the occurrence of a conduction disturbance through the “distal” or “peripheral” ramifications of the right bundle branch; and (b) the slightly earlier activation of the left ventricular apical endocardium (in reference to the right ventricular apex) may have been due to an earlier emergence from the divisions of the, left bundle branch, presumably due to the greater length of the right bundle branch. In beats with preexcitation: (a) the “incomplete” right bundle branch block pattern was concealed because the right ventricular inflow tract was activated before the right ventricular apex; (b) the deltaright ventricular apical intervals were shorter than those of adults with Wolff-Parkinson-White type B; and (c) arrival of excitation at the left ventricular endocardium was a function, either of the impulse emerging from the left bundle branch, or of that propagating from the preexcited site. Therefore, the delta-left ventricular apical endocardial intervals were considered to have represented conduction time from preexcited region to endocardium of left ventricle only when it could be proven that the conduction time (from atrial site of origin to left ventricular apical endocardium) was shorter through the right sided accessory pathway than through the normal pathway. This study suggests that some beats, which may be interpreted as representing “pure” Wolff-Parkinson-White type B complexes from epicardial maps, may in reality be “fusion” complexes.</ce:simple-para></ce:abstract-sec></ce:abstract></head><tail><ce:bibliography><ce:section-title>References</ce:section-title><ce:bibliography-sec><ce:bib-reference id="bib1"><ce:label>1.</ce:label><sb:reference><sb:contribution langtype="en"><sb:authors><sb:author><ce:surname>Latour</ce:surname><ce:given-name>H</ce:given-name></sb:author><sb:author><ce:surname>Puech</ce:surname><ce:given-name>P</ce:given-name></sb:author></sb:authors></sb:contribution><sb:host><sb:edited-book><sb:book-series><sb:series><sb:title><sb:maintitle>Electrocardiographic Endocativitaire</sb:maintitle></sb:title></sb:series></sb:book-series><sb:date>1957</sb:date><sb:publisher><sb:name>Masson et 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Application to conduction velocity and anomalous A-V excitation</sb:maintitle></sb:title></sb:contribution><sb:host><sb:issue><sb:series><sb:title><sb:maintitle>Circulation</sb:maintitle></sb:title><sb:volume-nr>31</sb:volume-nr></sb:series><sb:date>1965</sb:date></sb:issue><sb:pages><sb:first-page>651</sb:first-page></sb:pages></sb:host></sb:reference></ce:bib-reference></ce:bibliography-sec></ce:bibliography></tail></article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Arrival of excitation at the left ventricular apical endocardium in Wolff-Parkinson-White syndrome type B</title></titleInfo><titleInfo type="alternative" lang="en" contentType="CDATA"><title>Arrival of excitation at the left ventricular apical endocardium in Wolff-Parkinson-White syndrome type B</title></titleInfo><name type="personal"><namePart type="given">Otto L.</namePart><namePart type="family">Garcia</namePart><namePart type="termsOfAddress">M.D.</namePart><description>Associate Professor of Pediatrics</description><description>From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Agustin</namePart><namePart type="family">Castellanos</namePart><namePart type="termsOfAddress">M.D.</namePart><description>Professor of Medicine</description><description>From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Maria C.</namePart><namePart type="family">Vagueiro</namePart><namePart type="termsOfAddress">M.D.</namePart><description>Associate, Professor of Medicine</description><description>From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Robert J.</namePart><namePart type="family">Myerburg</namePart><namePart type="termsOfAddress">M.D.</namePart><description>Professor of Medicine</description><description>From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Henry</namePart><namePart type="family">Gelband</namePart><namePart type="termsOfAddress">M.D.</namePart><description>Professor of Pediatrics</description><description>From the University of Miami School of Medicine, Miami,Florida, U.S.A.; and the University of Lisbon, Lisbon, Portugal.</description><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="Full-length article"></genre><originInfo><publisher>ELSEVIER</publisher><dateIssued encoding="w3cdtf">1982</dateIssued><copyrightDate encoding="w3cdtf">1982</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">Electrograms were recorded from the His bundle area, right ventricular apex, right ventricular inflow tract, and left ventricular apical endocardium, in four patients (aged, 1, 1, 1.5, and 16 years) with Wolff-Parkinson-White syndrome type B. In beats without preexcitation: (a) delayed activation of the right ventricular inflow tract reflected the occurrence of a conduction disturbance through the “distal” or “peripheral” ramifications of the right bundle branch; and (b) the slightly earlier activation of the left ventricular apical endocardium (in reference to the right ventricular apex) may have been due to an earlier emergence from the divisions of the, left bundle branch, presumably due to the greater length of the right bundle branch. In beats with preexcitation: (a) the “incomplete” right bundle branch block pattern was concealed because the right ventricular inflow tract was activated before the right ventricular apex; (b) the deltaright ventricular apical intervals were shorter than those of adults with Wolff-Parkinson-White type B; and (c) arrival of excitation at the left ventricular endocardium was a function, either of the impulse emerging from the left bundle branch, or of that propagating from the preexcited site. Therefore, the delta-left ventricular apical endocardial intervals were considered to have represented conduction time from preexcited region to endocardium of left ventricle only when it could be proven that the conduction time (from atrial site of origin to left ventricular apical endocardium) was shorter through the right sided accessory pathway than through the normal pathway. This study suggests that some beats, which may be interpreted as representing “pure” Wolff-Parkinson-White type B complexes from epicardial maps, may in reality be “fusion” complexes.</abstract><note>The costs of publication of this article were defrayed in part bythe payment of page charges. This article must therefore be hereby marked “advertisement” in accordance with 18 U.S.C. § 1734 solely to indicate this fact.</note><relatedItem type="host"><titleInfo><title>Journal of Electrocardiology</title></titleInfo><titleInfo type="abbreviated"><title>YJELC</title></titleInfo><genre type="Journal">journal</genre><originInfo><dateIssued encoding="w3cdtf">1982</dateIssued></originInfo><identifier type="ISSN">0022-0736</identifier><identifier type="PII">S0022-0736(82)X8001-5</identifier><part><detail type="volume"><number>15</number><caption>vol.</caption></detail><detail type="issue"><number>2</number><caption>no.</caption></detail><extent unit="issue pages"><start>103</start><end>204</end></extent><extent unit="pages"><start>165</start><end>172</end></extent></part></relatedItem><identifier type="istex">8849C84861D8758EDCCBACC7D0692B08F77FF1C1</identifier><identifier type="DOI">10.1016/S0022-0736(82)80012-5</identifier><identifier type="PII">S0022-0736(82)80012-5</identifier><identifier type="ArticleID">82800125</identifier><accessCondition type="use and reproduction" contentType="">© 1982Research in Electrocardiology, Inc.</accessCondition><recordInfo><recordContentSource>ELSEVIER</recordContentSource><recordOrigin>Research in Electrocardiology, Inc., ©1982</recordOrigin></recordInfo></mods></metadata><enrichments><istex:catWosTEI uri="https://api.istex.fr/document/8849C84861D8758EDCCBACC7D0692B08F77FF1C1/enrichments/catWos"><teiHeader><profileDesc><textClass><classCode scheme="WOS">CARDIAC & CARDIOVASCULAR SYSTEMS</classCode></textClass></profileDesc></teiHeader></istex:catWosTEI></enrichments><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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